{"created":"2023-06-20T15:52:52.401247+00:00","id":7233,"links":{},"metadata":{"_buckets":{"deposit":"acf789ff-2a1e-40c0-884f-cc378edf91f8"},"_deposit":{"created_by":34,"id":"7233","owners":[34],"pid":{"revision_id":0,"type":"depid","value":"7233"},"status":"published"},"_oai":{"id":"oai:redcross.repo.nii.ac.jp:00007233","sets":["195:197:199"]},"author_link":["26040","26041","26039"],"item_10002_biblio_info_7":{"attribute_name":"書誌情報","attribute_value_mlt":[{"bibliographicIssueDates":{"bibliographicIssueDate":"2010-12","bibliographicIssueDateType":"Issued"},"bibliographicIssueNumber":"1","bibliographicPageEnd":"24","bibliographicPageStart":"17","bibliographicVolumeNumber":"35","bibliographic_titles":[{"bibliographic_title":"松山赤十字病院医学雑誌"}]}]},"item_10002_description_5":{"attribute_name":"抄録","attribute_value_mlt":[{"subitem_description":"IgG4関連疾患という概念が広く認識され始めたのは、ここ数年のことである。悪性腫瘍と鑑別を要するような結節性病変を形成するが、炎症性・反応性の疾患と考えられており、ステロイド療法で軽快するため、的確な診断が望まれる。全身のあらゆる臓器に発生し、腫瘤を形成する。病理組織学的にはリンパ濾胞を伴う多数の炎症細胞浸潤が見られ、強い線維化を伴う。特に形質細胞の浸潤が強く、IgGのサブタイプであるIgG4を産生するのが特徴的である。血清IgG4は高値となり、免疫組織化学染色でも陽性細胞を確認できる。当院での症例が蓄積されてきたため、それらの臨床病理学的特徴をここに報告する。(著者抄録)","subitem_description_type":"Abstract"}]},"item_10002_source_id_11":{"attribute_name":"書誌レコードID","attribute_value_mlt":[{}]},"item_10002_source_id_9":{"attribute_name":"ISSN","attribute_value_mlt":[{"subitem_source_identifier":"0385-3888","subitem_source_identifier_type":"ISSN"}]},"item_10002_version_type_20":{"attribute_name":"著者版フラグ","attribute_value_mlt":[{"subitem_version_resource":"http://purl.org/coar/version/c_970fb48d4fbd8a85","subitem_version_type":"VoR"}]},"item_creator":{"attribute_name":"著者","attribute_type":"creator","attribute_value_mlt":[{"creatorNames":[{"creatorName":"飛田, 陽"}],"nameIdentifiers":[{}]},{"creatorNames":[{"creatorName":"水野, 洋輔"}],"nameIdentifiers":[{}]},{"creatorNames":[{"creatorName":"大城, 由美"}],"nameIdentifiers":[{}]}]},"item_files":{"attribute_name":"ファイル情報","attribute_type":"file","attribute_value_mlt":[{"accessrole":"open_date","date":[{"dateType":"Available","dateValue":"2015-03-30"}],"displaytype":"simple","filename":"飛田先生-144.pdf","filesize":[{"value":"1.6 MB"}],"format":"application/pdf","licensetype":"license_note","mimetype":"application/pdf","url":{"label":"matsuyama00035","url":"https://redcross.repo.nii.ac.jp/record/7233/files/飛田先生-144.pdf"},"version_id":"bce32ce8-6b6e-4527-9635-dcf85f474589"}]},"item_keyword":{"attribute_name":"キーワード","attribute_value_mlt":[{"subitem_subject":"IgG4-related disease","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"IgG4-related sclerosing disease","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"autoimmune pancreatitis","subitem_subject_language":"en","subitem_subject_scheme":"Other"},{"subitem_subject":"Mikulicz's disease","subitem_subject_language":"en","subitem_subject_scheme":"Other"}]},"item_language":{"attribute_name":"言語","attribute_value_mlt":[{"subitem_language":"jpn"}]},"item_resource_type":{"attribute_name":"資源タイプ","attribute_value_mlt":[{"resourcetype":"departmental bulletin paper","resourceuri":"http://purl.org/coar/resource_type/c_6501"}]},"item_title":"当院におけるIgG4関連疾患の臨床病理学的検討","item_titles":{"attribute_name":"タイトル","attribute_value_mlt":[{"subitem_title":"当院におけるIgG4関連疾患の臨床病理学的検討"}]},"item_type_id":"10002","owner":"34","path":["199"],"pubdate":{"attribute_name":"公開日","attribute_value":"2015-03-30"},"publish_date":"2015-03-30","publish_status":"0","recid":"7233","relation_version_is_last":true,"title":["当院におけるIgG4関連疾患の臨床病理学的検討"],"weko_creator_id":"34","weko_shared_id":-1},"updated":"2023-06-20T20:14:25.204477+00:00"}